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Its potential application in managing gut inflammation has garnered significant attention due to its promising results in preclinical studies

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However, in resistant patients, oxidative stress causes a conformational change in Keap1, leading to the release of NRF2, which translocates to the nucleus and binds to antioxidant response element (ARE), activating the expression of downstream antioxidant genes, including GPX4, NQO1, HO-1 and SOD1 [33], and promoting the synthesis of GSH, as illustrated in Fig

Consequently, intracellular GSH depletion increases the free copper pool, promotes protein oligomerization, and heightens sensitivity to copper-mediated cell death [110]