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The broader neuroprotective profile of BPC-157 further supports its relevance

AA in its reduced state acts as the cofactor of violaxanthine de-epoxidase and maintains the dissipation of the excess excitation energy (Smirnoff, 2000)

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Collectively, these results indicate that -KG availability can be dictated by different metabolic enzymes depending on the cellular contexts, while -KG insufficiency broadly reduces AMPK expression and compromises energy sensing