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Laboratory protocols demonstrate that under optimal dosing conditions, the peptide eliminates senescent cells while destroying an undetectable number of healthy cells, representing exceptional selectivity compared to earlier senolytic approaches

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Antioxidant and Oxidative Stress Pathways A recurring theme across Epitalon cellular aging studies is the peptides proposed interaction with cellular antioxidant defence systems, a pathway also explored through the GHK-Cu Peptide , for research including: Superoxide dismutase (SOD) activity Catalase and glutathione peroxidase expression Reduction of lipid peroxidation markers in laboratory models These pathways are relevant because oxidative stress accumulation is considered a fundamental contributor to cellular ageing across multiple experimental models

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