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This beneficial effect was supposedly due to the increased expression of genes involved in biotic stress response ( PR1 ), ethylene biosynthesis and ROS scavenging including a GST (Tiwari et al., P

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Ferroptosis is morphologically, biochemically, and genetically distinct from necrosis, apoptosis, and autophagy, and these differing features include abnormal mitochondrial membrane density, iron accumulation, lipid peroxidation, overexpression of ferroptosis biomarkers, and death of leucocyte subsets and the corresponding loss of immune function [11, 18,19,20,21]

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