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These differences were recently revealed to have a fundamental impact on the outcome of antagonist engagement of FcRn, as a direct comparison of recombinant versions of efgartigimod (ARGX-113) and two other pipeline antagonistsbatoclimab (HL161BK) and nipocalimab (N027)revealed differential effects on the subcellular trafficking of FcRn [237]

It results in thick, silvery plaques caused by uncontrolled proliferation of keratinocytes

This, in turn, doubles the capacity of the available growth factors to promote signaling cascades necessary for mitogenesis (cell division), motogenesis (promotion of cellular motility), morphogenesis (structural development), neurogenesis (growth and development of nervous tissue), production of stem cells, and protection of a wide range of cells against injury