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In conclusion, the present study analysed the potential of CysLTR1 blockade in asthma pathogenesis, indicating that it could effectively suppress inflammation, airway remodeling, bronchial epithelial cell apoptosis and oxidative stress to improve asthma, potentially by activating the Nrf2 signaling pathway

Dry eye syndrome in thyroid-associated ophthalmopathy: lacrimal expression of TSH receptor suggests involvement of TSHR-specific autoantibodies

Significance of glutathione peroxidase 4 and intracellular iron level in ovarian cancer cells-"utilization" of ferroptosis mechanism

Does membrane fluidity become restricted, leading to the failure of membrane-related functions, or does the structural disruption of the membrane lead to ferroptosis due to membrane rupture