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Preclinical studies explore how the peptide behaves in in vitro environments and animal models, examining factors such as cellular communication, inflammatory signaling, vascular-related pathways, and structural tissue responses

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Although the exact epigenetic mechanisms directly involved in the pathogenesis of endometriosis are not yet known, evidence suggests that endometriosis could be associated with polyepigenetic alterations involving major signaling pathways (steroid hormone signaling, development, immunity, cell adhesion, Wnt- and MAPK-signaling, etc.)

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