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N-acetyl-cysteine administration supplies the cysteine necessary for GSH synthesis and concomitantly reduces hyperhomocysteinemia, improving GSH peroxidase activity and reducing oxidative stress (159)

151 While preclinical/clinical studies suggest that the dual/pan-PPAR agonists have a more significant effect in the treatment of disease when compared with PPAR or PPAR agonists alone, improvement of the low agonistic activity and low metabolic stability of multiple agonists may still need to be improved