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Targeting receptor-mediated endocytotic pathways with nanoparticles: rationale and advances

In IPF, environmental factors such as cigarette smoke, air pollution, and microbial infections damage alveolar epithelial cells (AECs) and microvascular endothelial cells, initiating an aberrant injuryrepair cascade [33,34,35]

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These structural modifications are designed to reduce interaction with IGF-binding proteins and increase peptide persistence and receptor engagement in controlled experimental systems, making it a more stable investigative tool relative to native IGF-1 in laboratory research applications