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Such alterations were also associated with inhibition of cytochrome c oxidase (CcOX) activity and microtubule network disassembly, which are considered hallmarks of nitric oxide (NO) toxicity, indicating that NO, rather than the depletion of GSH per se, is the primary mediator of cell damage
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It is important to mention that mitochondrial protection in astrocytes is an important asset to maintain the energetic balance of the brain and the antioxidant production that contribute to neuronal protection
Among those defects, treatment of intrabony defects has been studied and very well researched with randomized controlled clinical trials and long-term studies [16,17,18,19,20,21]
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