The mechanisms that may contribute to the induction of oxidative damage in diabetic patients include hyperglycemia-induced glucose auto-oxidation, non-enzymatic glycation of proteins and lipids, increased sorbitol pathway activity, oxidation of AGEs, and formation of prostaglandin H2 (PGH2) [311]
As a free radical, it can modify numerous cellular components, as described above (Lushchak and Lushchak, 2021)
They work best when paired with: Rehab that respects the biology of tissue healing Movement strategies that offload stress and restore coordination Patient education that reframes expectations You cant inject your way out of bad movement
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