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Biochem J (2001) 360:55762

Tsai, et al

It is hypothesized that eryptosis in patients with HB-ACLF is, at least in part, stimulated by specific components in the plasma, causing accelerated eryptosis and contributing to the development of anemia [250]

As AAs can modulate mTORC1 activity via multiple mechanisms, which involve receptors and transceptors that specifically recognize different types of AAs, it is likely that the modulation of mTORC1 activity is different for each type of AA