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Mycotoxins generate reactive oxygen species (ROS) and deplete antioxidant reserves, contributing to cellular damage and mitochondrial dysfunction 32

This attribute is particularly beneficial for individuals with genetic conditions affecting glutathione synthetase, as adding S-Acetyl-L-glutathione to fibroblast cultures has been shown to effectively restore GSH levels

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3) regulates the mitochondrial apoptotic pathway, reduces the release of Cyt c and cleaved caspase-3, increases Bcl-2 and hypoxia-inducible factor 1-alpha (HIF1), and inhibits oocytes and GC apoptosis