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Mitochondrial abnormalities and synaptic damage in huntingtons disease: A focus on defective mitophagy and Mitochondria-Targeted therapeutics

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Its critical for: Energy production Converting food into usable cellular fuel DNA repair Protecting against aging and cellular damage Metabolic function Regulating blood sugar, fat metabolism, and weight Cognitive clarity Supporting focus, memory, and mental sharpness Longevity pathways Activating sirtuins, which influence healthy aging The NAD+ Decline Problem NAD+ levels decline by up to 50% between ages 40 and 60

M., Galal, E.-T