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In advanced CKD, lipoproteins are more likely to undergo further oxidation to become ox-LDL, which is a potent proinflammatory agent and is highly correlated with artherosclerosis pathogenesis [38, 39]

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Presented data may be of advantage not only when comparing glutamate and GABA homeostasis and neurotransmission, but also for glutamine reserve display as neurotransmitter precursor and ammonia transfer buffer in glutamate/GABA/glutamine cycle within the intact brain substructures as well after ischemic insult in rats