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Impaired insulin signaling, diminished mitochondrial oxidative capacity, lipid peroxidation, ferroptosis, and microbial dysbiosis not only exacerbate hepatic steatosis but also facilitate the progression from simple steatosis to more severe forms of the disease, such as NASH and cirrhosis [51,56,63,64,65,66,67]

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Patel, S., Shende, S., Arora, S., Singh, A., Rastogi, S., & Rawat, A

The limitations include resistant anatomical sites such as fingers, toes, and bony prominences and areas lacking a melanocyte reservoir, such as segmental lesions with leucotrichia