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Interestingly, ApoE4 carriers have lower LTF levels in their saliva, which would serve as another mechanistic connection between ApoE4 and AD pathogenesis (Carro et al., 2017)

Conclusion: Previously reported LAAS-related genetic variants may have an indirect effect on sICAS pathogenesis, e.g., rs112735431 associated with a spectrum of stroke risk factors and rs2107595 polymorphism correlated with stroke mechanisms

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