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Collectively, the gut microbiota profile of the VPA-ASD groupmarked by an elevated Firmicutes/Bacteroidota ratio and an increase in pathobionts like Desulfovibrionalesaligns with established dysbiotic features of ASD

pylori infection by modulating the gastric innate immune response, significantly reducing the levels of TNF-, IL-8, and Monocyte Chemotactic Protein 1 (MCP-1) in the gastric mucosa of infected mice, while increasing the expression of Interferon-gamma (IFN-) and IL-10 (Garcia-Castillo et al., 2020)

Consequently, a decrease in Cu availability in these cells due to a decrease in metallothionein production and Cu uptake in the intestine would result in a decrease in dopamine degradation by the MAO pathway, since the activity of this enzyme is Cu-dependent (154)

Jeong, H